The High Serum Cholesterol Myth

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The High Serum Cholesterol Myth

Physicians used to think we only had to worry about our total cholesterol levels.

Then research found this wasn’t a strong predictor of heart disease.

The most important drug (aspirin) in preventing heart attacks had no effect on reducing cholesterol (but it does a great job of reducing inflammation).

Today lowering cholesterol is the #1 priority of every cardiologist in America.

Studies found increased serum cholesterol levels occurred more often in people with heart disease.

But the increase was 5-10% higher in those who developed heart disease than those who didn’t.

A study in Europe confirmed this lack of linkage between high cholesterol and death from heart attacks.

As heart disease researchers found their serum cholesterol links getting weaker and weaker, they came up with more complex scenarios.

Since total cholesterol wasn’t found to be very predicative of cardiovascular death, then perhaps part of total cholesterol was responsible.

The “bad” cholesterol, found in the low-density lipoprotein (LDL) particles, was now the prime culprit.

This launched a new war against “bad” cholesterol.

The story of “bad” cholesterol got even more complex as researchers discovered 2 types of LDL particles.

One type consists of large, fluffy LDL particles (good “bad” cholesterol) appearing to have little potential to cause the development of plaques on arteries.

The other type consists of small, dense LDL particles (bad “bad” cholesterol) strongly associated with an increased risk of heart disease.

So now you can have both good “bad” cholesterol (large fluffy LDL particles) and bad “bad” cholesterol (small dense LDL).


Well, so is everyone else fighting the cholesterol wars.

You very easily can identify whether you have the bad “bad” cholesterol or the good “bad” cholesterol.

All you need to do is find your ratio of triglycerides (TGs) to HDL cholesterol (TG/HDL), which will be listed on the results of your last fasting cholesterol screening.

If your ratio is less than 2, you have predominantly large, fluffy LDL particles that aren’t going to do you much harm.

If your ratio is greater than 4, you have a lot of small, dense LDL particles which can accelerate the development of plaques.

The connection between the TG/HDL ratio and heart attacks was confirmed by studies from Harvard Medical School.

This research found the higher your TG/HDL ratio, the higher your risk of having a heart attack.

Those with the highest TG/HDL ratio had 16 times the risk compared to those with the lowest ratio.

That’s a huge increase in risk for the most common cause of death!

A high TG/HDL ratio does far more than increase your risk of heart disease.

It’s a marker of a metabolic syndrome, which shows you’re developing insulin resistance.

Insulin resistance leads to obesity, type 2 diabetes, and eventually to accelerated heart disease.

As your TG/HDL ratio increases, this means your insulin resistance is also increasing.

The consequence of increased insulin levels means your body is churning out way too much AA.

The more AA you produce, the more silent inflammation you generate.

Another study found people with a low TG/HDL ratio who smoked, didn’t exercise, and had hypertension and elevated levels of LDL cholesterol had about a 50% lower risk of developing heart disease than those who had a far better lifestyle but a higher TG/HDL ratio.

This means lowering your TG/HDL ratio may have a far greater impact on whether you develop heart disease than adopting these improved lifestyle factors.

Does this mean you should smoke, stay sedentary, and not worry about your blood pressure or cholesterol levels?

No, but it means you need to lower your TB/HDL ratio if your goal is to reduce heart disease.

Come join us on our natural weight loss journey!  We’d love to have you along!

Have an awesome day!

(Based on Dr. Barry Sears’ “The Anti-Inflammation Zone”)

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Dick and Lenay

email: – 715-431-0657

P.S. If your diet isn’t working for you, join us on our natural weight loss journey.


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